{"count":159558,"next":"https://api-test.medunigraz.at/v1/research/publication/?format=json&limit=20&offset=158200&ordering=impactfactor_norm","previous":"https://api-test.medunigraz.at/v1/research/publication/?format=json&limit=20&offset=158160&ordering=impactfactor_norm","results":[{"id":211964,"title":"In-host evolution of Burkholderia pseudomallei.","abstract":"Burkholderia pseudomallei is a gram-negative rod-shaped bacterium responsible for the severe disease melioidosis. B. pseudomallei has the capability to infect both non-phagocytic and phagocytic cells and has evolved several mechanisms to evade the host immune system. A previous study identified a B. pseudomallei isolate, which showed altered survival within macrophages compared to its isogenic wildtype after passage through human macrophages. The aim of this study was to further characterize the virulence of this isolate (E8_evolved) and to elucidate the underlying mechanisms. B. pseudomallei E8_evolved exhibited increased intracellular invasion and cytotoxicity compared to the isogenic wildtype. This suggests a potentially increased invasion or improved phagosomal escape, potentially leading to a higher bacterial load early in the infection. To determine whether pyroptosis is responsible for the observed increase in cytotoxicity, western blot analyses were performed. The cleavage product of gasdermin D, an indicator of pyroptosis, was detected at a similar intensity in E8_evolved and the wildtype strain. Next caspase-1 and caspase-4, which activate gasdermin D during canonical or non-canonical inflammasome activation, were investigated. While caspase-1 cleavage was similar in both strains, the caspase-4 cleavage product showed increased intensity in E8_evolved indicating increased activation. As no phosphorylated MLKL was detected, necroptosis can be excluded as a potential cause of cell lysis. Additionally, it was observed that the increased cytotoxicity is not associated with elevated production and secretion of the cytokines TNF-α, IL-1α, and IL-1β. Specifically, the concentration of the cytokine TNF-α is reduced in the mutant strain three hours post-infection. To explore changes at the DNA level, a whole genome sequencing analysis of the mutant strain was conducted. This analysis revealed two mutations in the potential T3SS regulatory protein HpaA, specifically in the amino acids VAL25 and ARG28. However, since this protein remains uncharacterized, further research is required to fully understand its role and validate the potential impact of these mutations. Taken together, the mutant exhibits enhanced invasion and bacterial loads, potentially attributable to mutations in the putative T3SS regulatory protein HpaA.","authors":["Harlander, J"],"year":2024,"source":" [ Diplomarbeit/Master Thesis (UNI) ] Universität Graz; 2024. pp.57. 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","category":1,"document_type":1,"sci":null,"pubmed":null,"doi":"10.1556/EuJMI.2.2012.1.10","pmc":null,"organizations":["122070-14020"],"persons":["122070-50496"],"imported":"2012-10-09T17:00:54+02:00","journal":null,"issn":"2062-509X","collection_publisher":null,"collection_title":null,"edition":null,"university":null,"country":null,"case_report":false,"impactfactor":null,"impactfactor_year":null,"impactfactor_norm":null,"impactfactor_norm_year":null,"impactfactor_norm_category":null,"impactfactor_norm_super":null,"impactfactor_norm_super_year":null,"impactfactor_norm_super_category":null,"citations":null,"conference_name":null,"conference_place":null,"conference_international":false,"scientific_event":false,"invited_lecture":false,"keynote_speaker":false,"selected_presentation":false,"biobank_use":false,"bmf_use":false,"zmf_use":false,"local_affiliation":true},{"id":218675,"title":"Innovative Versorgungsmöglichkeiten – der Blick nach Österreich","abstract":null,"authors":["Jagsch, C","Bittelmayer, S","Glarcher, M"],"year":2025,"source":" In: Horneber, M; Püllen R. (Hrsg);  editors(s). Das demenzsensible Krankenhaus\r\nGrundlagen und Praxis einer patientenorientierten Betreuung und Versorgung. 1: Stuttgart: Kohlhammer;  2025(ISBN: ISBN 978-3-17-044655) ","category":6,"document_type":null,"sci":null,"pubmed":null,"doi":null,"pmc":null,"organizations":["218675-29484"],"persons":["218675-71002"],"imported":"2025-11-18T12:09:54+01:00","journal":null,"issn":null,"collection_publisher":"Horneber, M; Püllen R. (Hrsg); ","collection_title":"Das demenzsensible Krankenhaus\r\nGrundlagen und Praxis einer patientenorientierten Betreuung und Versorgung","edition":null,"university":null,"country":null,"case_report":false,"impactfactor":null,"impactfactor_year":null,"impactfactor_norm":null,"impactfactor_norm_year":null,"impactfactor_norm_category":null,"impactfactor_norm_super":null,"impactfactor_norm_super_year":null,"impactfactor_norm_super_category":null,"citations":null,"conference_name":null,"conference_place":null,"conference_international":false,"scientific_event":false,"invited_lecture":false,"keynote_speaker":false,"selected_presentation":false,"biobank_use":false,"bmf_use":false,"zmf_use":false,"local_affiliation":true},{"id":218677,"title":"Hochrisikogruppe ältere Männer - Möglichkeiten und Grenzen zielgerichteter Suizidprävention\r\n","abstract":null,"authors":["Jagsch, C"],"year":2025,"source":"Fachtagung Suizidprävention 2025 Kärnten\r\n; 9. Sept. 2025; Klagenfurt. 2025. ","category":3,"document_type":null,"sci":null,"pubmed":null,"doi":null,"pmc":null,"organizations":["218677-29484"],"persons":["218677-71002"],"imported":"2025-11-18T12:19:27+01:00","journal":null,"issn":null,"collection_publisher":null,"collection_title":null,"edition":null,"university":null,"country":null,"case_report":false,"impactfactor":null,"impactfactor_year":null,"impactfactor_norm":null,"impactfactor_norm_year":null,"impactfactor_norm_category":null,"impactfactor_norm_super":null,"impactfactor_norm_super_year":null,"impactfactor_norm_super_category":null,"citations":null,"conference_name":true,"conference_place":true,"conference_international":false,"scientific_event":true,"invited_lecture":true,"keynote_speaker":false,"selected_presentation":false,"biobank_use":false,"bmf_use":false,"zmf_use":false,"local_affiliation":true},{"id":219670,"title":"Hereditary Cholestasis Syndromes and Their Clinical Appearance – Review of the literature and cases at the Medical University Graz","abstract":"Background: Hereditary cholestasis syndromes are a heterogeneous group of rare liver diseases that are caused by genetic defects in genes related with bile acid transport or metabolism. Although they are rare, these diseases are clinically important as affected patients can present with severe clinical symptoms like jaundice, pruritus as well as further complications like chronic liver disease, potentially requiring a liver transplant as ultimate therapy. Cholestatic pruritus can greatly reduce quality of life. With new treatments like ASBT inhibitors now available, it’s important to know how common this condition is in order to understand how many patients could benefit from these new options. Throughout this wide group of syndromes, clinical presentation can vary between types of genetic mutations, age groups, and triggering events such as infections, pregnancy or drug medications, possibly triggering the manifestation of symptoms in patients with a genetic predisposition. Methods: We retrospectively analyzed patients with a suspected hereditary cause of cholestasis who underwent genetic testing at our university hospital throughout the last 20 years. Clinical data, laboratory findings, and genetic variants (e.g. mutations, SNPs) were reviewed to evaluate genotype-phenotype correlations, clinical presentation by age, and potential disease triggers. Results: From medical records, we identified 88 patients, 64 children and 24 adults, with a possible hereditary cause of cholestasis. Only 53 patients presented with a clear cholestatic phenotype (such as PFIC, BRIC, ICP or LPAC). The remaining 35 patients had evidence of cholestasis or mutations but without a final clinical classification at the time of research. Genetic variants were identified in 50 out of 88 patients, most frequently in the ABCB11 and ABCB4 gene. 38 patients had mutations without a clear clinical diagnosis. The clinical presentation varied by age. Pediatric cases (predominantly male (44 out of 64)) were more often associated with ABCB11 variants and presented with jaundice, whereas adult patients (predominantly female (16 out of 24)) more often carried ABCB4 variants and presented with biliary symptoms or gallstone disease. Simply determination of zygosity status of a variant (i.e. homo- or heterozygote variants) cannot predict clinical appearance since remaining function of the protein a critical contributing factor. Pruritus was documented in only approximately 6% of cases at the time of diagnosis but in around 22% during disease course, clustering with ABCB11 variants in children and ABCB4 or ATP8B1 variants in adults. Treatment was mainly symptomatic, with UDCA prescribed in 32 patients and in only two cases a specific antipruritic therapy was documented. An unexpected finding was the occurrence of EBV infections as a potential trigger for cholestatic episodes in eight patients, five of whom had mutations in ABCB11.","authors":["Payam, E"],"year":2025,"source":"Humanmedizin; [ Diplomarbeit ] Medizinische Universität Graz; 2025. pp. 59","category":5,"document_type":15,"sci":null,"pubmed":null,"doi":null,"pmc":null,"organizations":[],"persons":[],"imported":"2026-01-07T11:07:20+01:00","journal":null,"issn":null,"collection_publisher":null,"collection_title":null,"edition":null,"university":"Medizinische Universität Graz","country":"40","case_report":false,"impactfactor":null,"impactfactor_year":null,"impactfactor_norm":null,"impactfactor_norm_year":null,"impactfactor_norm_category":null,"impactfactor_norm_super":null,"impactfactor_norm_super_year":null,"impactfactor_norm_super_category":null,"citations":null,"conference_name":null,"conference_place":null,"conference_international":false,"scientific_event":false,"invited_lecture":false,"keynote_speaker":false,"selected_presentation":false,"biobank_use":false,"bmf_use":false,"zmf_use":false,"local_affiliation":false},{"id":219655,"title":"Der Vergleich und die Limitationen unterschiedlicher Formeln zur QTc-Zeitberechnung im Kindes- und Jugendalter","abstract":"Hintergrund: Das QT-Intervall im Elektrokardiogramm (EKG) spiegelt die Gesamtdauer der ventrikulären Depolarisation und Repolarisation wider und stellt somit einen zentralen Parameter zur Beurteilung der kardialen Erregungsrückbildung dar. Aufgrund der Abhängigkeit des QT-Intervalls von der Herzfrequenz wird zur Beurteilung in der Regel die frequenzkorrigierte QT-Zeit (QTc) verwendet. Bisher besteht Uneinigkeit darüber, welche QTc-Formel am besten performt und daher zur QTc-Zeitbestimmung verwendet werden sollte. Besonders in der Pädiatrie treten aufgrund physiologischer Entwicklungsprozesse tendenziell höhere Herzfrequenzen auf, die die Ergebnisse der QTc-Berechnung beeinflussen können. Ziel dieser Arbeit war es daher, die QTc-Zeiten in einem pädiatrischen Kollektiv anhand verschiedener QTc-Formeln (Bazett, Fridericia, Framingham, Goto, Mayeda, Rautaharju-a, Kawataki, Wernicke) zu berechnen und deren Verhalten hinsichtlich einer Abhängigkeit von Herzfrequenz und Geschlecht zu vergleichen.","authors":["Musiol, C"],"year":2025,"source":"Humanmedizin; [ Diplomarbeit ] Medizinische Universität Graz; 2025. pp. 101","category":5,"document_type":15,"sci":null,"pubmed":null,"doi":null,"pmc":null,"organizations":[],"persons":[],"imported":"2026-01-07T11:07:20+01:00","journal":null,"issn":null,"collection_publisher":null,"collection_title":null,"edition":null,"university":"Medizinische Universität Graz","country":"40","case_report":false,"impactfactor":null,"impactfactor_year":null,"impactfactor_norm":null,"impactfactor_norm_year":null,"impactfactor_norm_category":null,"impactfactor_norm_super":null,"impactfactor_norm_super_year":null,"impactfactor_norm_super_category":null,"citations":null,"conference_name":null,"conference_place":null,"conference_international":false,"scientific_event":false,"invited_lecture":false,"keynote_speaker":false,"selected_presentation":false,"biobank_use":false,"bmf_use":false,"zmf_use":false,"local_affiliation":false},{"id":67270,"title":"F18-FDG for Staging Extrathoracal Lymph-Node Manifestations in Sarcoidosis","abstract":null,"authors":["Prager E, Bisail B, Schwarz T, Aigner RM"],"year":2007,"source":"ÖGN Kongreß; 45(35):A159-A159.-ÖGN Kongress-Jahrestagung der Nuklearmedizin/ 6. nationales Symposium; Jan, 25-27, 2007; Linz, AUSTRIA.","category":2,"document_type":9,"sci":null,"pubmed":null,"doi":null,"pmc":null,"organizations":["67270-14107"],"persons":["67270-51727","67270-50727"],"imported":"2007-05-22T10:09:42+02:00","journal":null,"issn":"0029-5566","collection_publisher":null,"collection_title":null,"edition":null,"university":null,"country":null,"case_report":false,"impactfactor":null,"impactfactor_year":null,"impactfactor_norm":null,"impactfactor_norm_year":null,"impactfactor_norm_category":null,"impactfactor_norm_super":null,"impactfactor_norm_super_year":null,"impactfactor_norm_super_category":null,"citations":null,"conference_name":true,"conference_place":true,"conference_international":false,"scientific_event":true,"invited_lecture":false,"keynote_speaker":false,"selected_presentation":true,"biobank_use":false,"bmf_use":false,"zmf_use":true,"local_affiliation":true},{"id":218937,"title":"Environmental footprint of patient travel: insights from a German clinical study","abstract":null,"authors":["Schunk, V","Gerhalter T"],"year":2025,"source":"Proceedings of the 30th Annual Congress of the World Muscle Society. 2025; -WMS 2025; OCT 7-11, 2025; Vienna, AUSTRIA. 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